Magnesium Glycinate in Dysmenorrhea: Mechanism in Reducing Prostaglandin F2-alpha
Magnesium is an essential enzymatic cofactor in over 300 biochemical reactions, including muscle contraction, neuromuscular conduction, and nitric oxide synthesis.
Clinical trials show that supplementing with 300–400mg of elemental magnesium glycinate throughout the luteal phase reduces baseline endometrial prostaglandin F2-alpha levels, significantly lowering the severity of menstrual cramps while enhancing restorative delta-wave sleep.
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